1.Cigarette smoking increases the irritability of the small airways, causes mucus hypersecretion, and decreases mucociliary transport.
吸烟增加小气道的反应性，使粘液分泌过多，降低粘膜-纤毛转运。
2.Mucus hypersecretion in airway is the common pathophyisolo gi cal characteristic of many chronic airway inflammation diseases.
目的：介绍慢性炎症气道粘液高分泌的治疗进展。
3.Objective To remedy lacrimal hypersecretion by procaine blocking lacrimal gland .
目的利用普罗卡因封闭泪腺治疗泪液分泌过多症。
4.AIM: To explore the mechanism of signal cascades in cigarette smoke-induced mucous hypersecretion.
目的：探讨香烟对气道黏液分泌的影响及其信号转导机制。
5.Objective To investigate the roles of theaflavins(TFs) in airway mucus hypersecretion induced by human neutrophil elastase(HNE).
目的观察茶黄素（TF）各单体在人中性粒细胞弹性蛋白酶（HNE）诱导气道黏液高分泌中的作用。
6.Mechanism of Chronic Airway Inflammation Mucous Hypersecretion Inhibited by Macrolides
大环内酯类抗生素对慢性气道炎症黏液高分泌的干预机制
7.Effect of Eucalyptus globulus oil on lipopolysaccharide-induced chronic bronchitis and mucin hypersecretion in rats
蓝桉油对脂多糖引起的大鼠慢性支气管炎及黏蛋白高分泌的影响
8.Molecular mechanism of mucus hypersecretion in chronic inflammatory airway diseases
慢性气道炎症黏液高分泌的分子机制
9.The Advance of Therapeutical Medication of Mucus Hypersecretion in Chronic Airway Inflammation
慢性炎症气道粘液高分泌治疗药物研究进展
10.Molecular mechanism of airway mucous hypersecretion inhibited by macrolides
大环内酯抗生素抑制气道粘液高分泌的分子机制